Ganesh Ujwal

Photosensitive epilepsies are characterized by visually-induced seizures. Flashing-light stimuli are known to induce seizures in some (but not all) patients. My question is whether people with this type of epilepsy can self-induce seizures by rapid eye blinking?

I have previously heard that, if there are multiple simultaneous painful stimuli, the mind will only feel the sensation of pain from the most severe injury. A large amount of googling has turned up nothing and PubMed searches are proving tricky as I cant find the name of this effect (if it exists). If it does exist, is the mechanism solely effective for pain signals or would we for example only feel cold at our coldest points (i.e. from the face or the hands rather than both) on a frosty morning?

T-cells recognize the MHC molecules and body's own peptides. When it doesn't, it alarms the immune system. But do T-cells express MHC molecules ? If so, how are they using it? If not, what happens when a virus infects T-cells? (Yes, I am confused about HIV infection mechanism too. They escape from immune response by altering their genes and disrupting the MHC-peptide bonding. I can see that this can work in macrophages. But what is the situation in T-cells ?)

I understand that one can translate a nucleotide sequence and run PSI-BLAST on the protein (proteins if you take the 6 reading frames), but I'm looking for distant homology for bacterial small RNAs (typically 50-200 nucleotides long and noncoding). If there is no such resource, what are the main obstacles to this implementation?