luminogenics

Yes, then the acetate is bound up by CoA, which is further oxidized into CO2 through various redox reactions, which we know as the Krebs cycle.

That's a little unclear. Glucose transport where? I seem to remember glucose is almost immediately bound once it is absorbed into the blood stream.

Yes the proteins are for transcription factor that activate genes that would otherwise be dormant in a differentiated cell.

Also, A bacterium may have a chromosome and plasmids. So there is some distinction based on size of the molecule. In some cases the distinction between chromosome and plasmid gets a little fizzy, so like many other concepts it depends on what you mean.

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Yeah this all happen when the host cell has been exposed to environmental stress. Including UV light and starvation. In either of these cases internal levels of the repressor drop and you get activation. All of these promoters are leaky anyway, what is important is the volume and redundancy of promoter sites. In this case only when the cell has been starving for a couple hours, and finds itself in a condition of excess waste it's not going to be able to keep up the level of protein production to keep the virus from escaping. Same for UV light, even if the cell is growing fine, if it finds itself replicating mutations the promoter and repressor sites are going to change and the repressor won't fit as well, and again the virus pops out. I would suggest if you want to find a good article either look for a paper where they describe the structure of the repressor (Cell circa 2001) or, look back to the initial sequencing of the promoter region.