R2GH

I hope I can explain my question well enough since Im not a native speaker :) Basically I was wondering, why as in reaction to a diabetes I condition the muscle is breaking down its own protein to amino acids, which then are farther moved to the liver resulting in gluconeogenesis? It makes no sense to me because I don's see the necessity of the muscle doing so during Diabetes I. In a similiar condition such starvation I do understand the necessity, because the body actually lacks glucoses and eventually runs out of all other sources to possibly synthesise glucoses through gluconeogenesis und thus needs to resort to glucoplasmatic amino acids. However during diabetes I condition, there is enough glucoses in the blood stream so why is the muscle breaking down the protein ? Thanks for the help :)