Deslaar

Incorrect. Classical Chaotic Systems are NOT Quantum Chaotic Systems. The distinction between them is that the former is deterministic and the latter is not, however, both are chaotic.

Chaotic systems such as the weather and plate tectonics are chaotic at a classical level not at quantum level. QM has nought to do with chaos theory.

You said that GABA can have a enhancing or inhibitory effect on the AP. What is the mechanism that "decides" which effect to have?

Interesting. I might have to some more research on this. Thanks.

And what is the mechanism that makes the distinction? BTW just checked Ledoux's book The Synaptic Self, and he states that dendrites can release more than one neurotransmitter.

Because a neuron connects to more than one other neuron. However, if you molec bio book says that a particular nerve releases only one neurotransmitter then I confused. How old is it?

Sure do. Axon->Synapse->Dendrite. Axon releases neuro transmitters into synapse, dendrite recieves neurotransmitters through receptors, receptors release calcium etc into cell body that affects its AP. Am I being unclear?

Great, then you should be able to explain it to me. If dendites have only one type of neurotransmitter then how can GABA and Glutamate receptors work together to produce inhibition and enhancing effects on the AP? How can cortisol play it's global role?

I appreciate that however the neurochemical makeup of the synapse is key factor in whether a post synaptic neuron will fire or not.

I don't think so. The impulse is called an action potential and while it is binary (on/off) it is triggered by an analog mechanism composed of receptors for inhibitors and enhancers. There should in principle, be an array of receptors on dendrites that reflect there function. Having dendrites that respond to only one neurotransmitter would severly reduce brain plasticity and neurological functions and I don't see any reason why evolution should produce such a constrained architecture.

Most people are here are probably familiar with neurotransmitters and receptors. Monoamines, glutamate, hormones, etc act as inhibitors or enhancers in neuronal AP firing processes in the synaptic pathways of the brain. I have some questions about the distribution of neuroreceptors. Is it true that the dendrites of neurons have a wide variation in which receptors they possess? If so is this the mechanism that allows neurotransmitters to target specific of parts of the brain and specific synaptic connections. Is it accurate to say that any given synaptic connection is uniquely coded with a "receptor signature"? Thanks in advance.